GALNS polyclonal antibody
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More Files
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Specification
Product Description
Goat polyclonal antibody raised against synthetic peptide of GALNS.
Immunogen
A synthetic peptide corresponding to human GALNS.
Sequence
C-TTHNLEDHTKLP
Host
Goat
Theoretical MW (kDa)
58
Reactivity
Human
Form
Liquid
Purification
Antigen affinity purification
Concentration
0.5 mg/mL
Quality Control Testing
Antibody Reactive Against Synthetic Peptide.
Recommend Usage
ELISA (1:64000)
Western Blot (0.01-0.03 ug/mL)
The optimal working dilution should be determined by the end user.Storage Buffer
In Tris saline, pH 7.3 (0.5% BSA, 0.02% sodium azide)
Storage Instruction
Store at -20°C.
Aliquot to avoid repeated freezing and thawing.Note
This product contains sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.
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Applications
Western Blot (Tissue lysate)
GALNS polyclonal antibody (Cat # PAB7554) (0.01 ug/mL) staining of human bone marrow lysate (35 ug protein in RIPA buffer). Primary incubation was 1 hour. Detected by chemiluminescence.Enzyme-linked Immunoabsorbent Assay
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Gene Info — GALNS
Entrez GeneID
2588Protein Accession#
NP_000503.1Gene Name
GALNS
Gene Alias
FLJ17434, FLJ42844, FLJ98217, GALNAC6S, GAS, MPS4A
Gene Description
galactosamine (N-acetyl)-6-sulfate sulfatase
Omim ID
253000Gene Ontology
HyperlinkGene Summary
This gene encodes N-acetylgalactosamine-6-sulfatase which is a lysosomal exohydrolase required for the degradation of the glycosaminoglycans, keratan sulfate, and chondroitin 6-sulfate. Sequence alterations including point, missense and nonsense mutations, as well as those that affect splicing, result in a deficiency of this enzyme. Deficiencies of this enzyme lead to Morquio A syndrome, a lysosomal storage disorder. [provided by RefSeq
Other Designations
chondroitinase
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Interactome
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Pathway
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Disease
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Publication Reference
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Combining an autologous peripheral nervous system "bridge" and matrix modification by chondroitinase allows robust, functional regeneration beyond a hemisection lesion of the adult rat spinal cord.
Houle JD, Tom VJ, Mayes D, Wagoner G, Phillips N, Silver J.
Journal of Neuroscience 2006 Jul; 26(28):7405.
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Combining an autologous peripheral nervous system "bridge" and matrix modification by chondroitinase allows robust, functional regeneration beyond a hemisection lesion of the adult rat spinal cord.
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