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GIPR polyclonal antibody

  • Catalog # : PAB26302
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  • Specification
  • Product Description:
  • Rabbit polyclonal antibody raised against synthetic peptide of GIPR.
  • Immunogen:
  • A synthetic peptide corresponding to 17 amino acids at N-terminal extracellular domain of human GIPR.
  • Host:
  • Rabbit
  • Reactivity:
  • Bovine, Dog, Human
  • Specificity:
  • BLAST analysis of the peptide immunogen showed no homology with other human proteins, except GHRHR (100%), TG (41%).
  • Form:
  • Liquid
  • Purification:
  • Immunoaffinity chromatography
  • Recommend Usage:
  • Immunohistochemistry (Formalin/PFA-fixed paraffin-embedded sections) (16 ug/mL)
    The optimal working dilution should be determined by the end user.
  • Storage Buffer:
  • In PBS (0.09% sodium azide)
  • Storage Instruction:
  • Store at 4°C. For long term storage store at -80°C.
    Aliquot to avoid repeated freezing and thawing.
  • Note:
  • This product contains sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.
  • Applications
  • Immunohistochemistry (Formalin/PFA-fixed paraffin-embedded sections)
  • Immunohistochemistry (Formalin/PFA-fixed paraffin-embedded sections)
  • Immunohistochemistry (Formalin/PFA-fixed paraffin-embedded sections) of human pancreas tissue with GIPR polyclonal antibody (Cat # PAB26302). Immunohistochemistry of formalin-fixed, paraffin-embedded tissue after heat-induced antigen retrieval.
  • Application Image
  • Immunohistochemistry (Formalin/PFA-fixed paraffin-embedded sections)
  • Immunohistochemistry (Formalin/PFA-fixed paraffin-embedded sections)
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  • Gene Information
  • Entrez GeneID:
  • 2696
  • Gene Name:
  • GIPR
  • Gene Alias:
  • MGC126722
  • Gene Description:
  • gastric inhibitory polypeptide receptor
  • Gene Summary:
  • Gastric inhibitory polypeptide (GIP; MIM 137240), also called glucose-dependent insulinotropic polypeptide, is a 42-amino acid polypeptide synthesized by K cells of the duodenum and small intestine. It was originally identified as an activity in gut extracts that inhibited gastric acid secretion and gastrin release, but subsequently was demonstrated to stimulate insulin release potently in the presence of elevated glucose. The insulinotropic effect on pancreatic islet beta-cells was then recognized to be the principal physiologic action of GIP. Together with glucagon-like peptide-1, GIP is largely responsible for the secretion of insulin after eating. It is involved in several other facets of the anabolic response.[supplied by OMIM
  • Other Designations:
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