CACNA1G polyclonal antibody
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Specification
Product Description
Goat polyclonal antibody raised against synthetic peptide of CACNA1G.
Immunogen
A synthetic peptide corresponding to amino acids at internal region of human CACNA1G.
Sequence
C-AEEISKREDASGQLS
Host
Goat
Theoretical MW (kDa)
262, 260, 259
Specificity
This antibody is expected to recognize isoform 1 (NP_061496.2), 4 (NP_938199.1), 5 (NP_938194.1), 8 (NP_938198.1), 9 (NP_938192.1), 10 (NP_938200.1) and isoform 15 (NP_938407.1).
Form
Liquid
Purification
Antigen affinity purification
Concentration
0.5 mg/mL
Recommend Usage
ELISA (1:32000)
The optimal working dilution should be determined by the end user.Storage Buffer
In Tris saline, pH 7.3 (0.5% BSA, 0.02% sodium azide)
Storage Instruction
Store at -20°C.
Aliquot to avoid repeated freezing and thawing.Note
This product contains sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.
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Applications
Enzyme-linked Immunoabsorbent Assay
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Gene Info — CACNA1G
Entrez GeneID
8913Protein Accession#
NP_061496.2;NP_938192.1;NP_938194.1;NP_938198.1;NP_938199.1;NP_938200.1;NP_938407.1Gene Name
CACNA1G
Gene Alias
Ca(V)T.1, Cav3.1, MGC117234, NBR13
Gene Description
calcium channel, voltage-dependent, T type, alpha 1G subunit
Omim ID
604065Gene Ontology
HyperlinkGene Summary
Voltage-activated calcium channels can be distinguished based on their voltage-dependence, deactivation, and single-channel conductance. See MIM 601011. Low-voltage-activated calcium channels are referred to as 'T' type because their currents are both transient, owing to fast inactivation, and tiny, owing to small conductance. T-type channels are thought to be involved in pacemaker activity, low-threshold calcium spikes, neuronal oscillations and resonance, and rebound burst firing.[supplied by OMIM
Other Designations
calcium channel, voltage-dependent, T type, alpha-1G subunit|calcium channel, voltage-dependent, alpha 1G subunit|voltage-dependent calcium channel alpha 1G subunit|voltage-dependent calcium channel alpha 1G subunit, isoform 11
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Interactome
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Pathway
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Disease
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Publication Reference
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Genetic enhancement of thalamocortical network activity by elevating alpha 1g-mediated low-voltage-activated calcium current induces pure absence epilepsy.
Ernst WL, Zhang Y, Yoo JW, Ernst SJ, Noebels JL.
The Journal of Neuroscience 2009 Feb; 29(6):1615.
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Genetic enhancement of thalamocortical network activity by elevating alpha 1g-mediated low-voltage-activated calcium current induces pure absence epilepsy.
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