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SNRPN 293T Cell Transient Overexpression Lysate(Denatured)

  • Catalog # : H00006638-T01
  • Visit Frequency :
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  • Specification
  • Transfected Cell Line:
  • 293T
  • Plasmid:
  • pCMV-SNRPN full-length
  • Host:
  • Human
  • Theoretical MW (kDa):
  • 26.51
  • Quality Control Testing:
  • Transient overexpression cell lysate was tested with Anti-SNRPN antibody (H00006638-B01) by Western Blots.
    SDS-PAGE Gel
    QC Testing of H00006638-T01
    SNRPN transfected lysate
    Western Blot
    QC Testing of H00006638-T01
    Lane 1: SNRPN transfected lysate ( 26.51 KDa).
    Lane 2: Non-transfected lysate.
  • Storage Buffer:
  • 1X Sample Buffer (50 mM Tris-HCl, 2% SDS, 10% glycerol, 300 mM 2-mercaptoethanol, 0.01% Bromophenol blue)
  • Storage Instruction:
  • Store at -80°C. Aliquot to avoid repeated freezing and thawing.
  • Applications
  • Western Blot
  • Application Image
  • Western Blot
  • Gene Information
  • Entrez GeneID:
  • 6638
  • Gene Name:
  • Gene Alias:
  • DKFZp686C0927,DKFZp686M12165,DKFZp761I1912,DKFZp762N022,FLJ33569,FLJ36996,FLJ39265,HCERN3,MGC29886,PWCR,RT-LI,SM-D,SMN,SNRNP-N,SNURF-SNRPN
  • Gene Description:
  • small nuclear ribonucleoprotein polypeptide N
  • Gene Summary:
  • The protein encoded by this gene is one polypeptide of a small nuclear ribonucleoprotein complex and belongs to the snRNP SMB/SMN family. The protein plays a role in pre-mRNA processing, possibly tissue-specific alternative splicing events. Although individual snRNPs are believed to recognize specific nucleic acid sequences through RNA-RNA base pairing, the specific role of this family member is unknown. The protein arises from a bicistronic transcript that also encodes a protein identified as the SNRPN upstream reading frame (SNURF). Multiple transcription initiation sites have been identified and extensive alternative splicing occurs in the 5' untranslated region. Additional splice variants have been described but sequences for the complete transcripts have not been determined. The 5' UTR of this gene has been identified as an imprinting center. Alternative splicing or deletion caused by a translocation event in this paternally-expressed region is responsible for Angelman syndrome or Prader-Willi syndrome due to parental imprint switch failure. [provided by RefSeq
  • Other Designations:
  • OTTHUMP00000159463,SM protein N,tissue-specific splicing protein
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