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Last updated: 2016/11/27

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HEXB 293T Cell Transient Overexpression Lysate(Denatured)

  • Catalog # : H00003074-T01
  • Visit Frequency :
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  • Specification
  • Transfected Cell Line:
  • 293T
  • Plasmid:
  • pCMV-HEXB full-length
  • Host:
  • Human
  • Theoretical MW (kDa):
  • 63.1
  • Quality Control Testing:
  • Transient overexpression cell lysate was tested with Anti-HEXB antibody (H00003074-B01) by Western Blots.
    SDS-PAGE Gel
    QC Testing of H00003074-T01
    HEXB transfected lysate.
    Western Blot
    QC Testing of H00003074-T01
    Lane 1: HEXB transfected lysate ( 63.1 KDa)
    Lane 2: Non-transfected lysate.
  • Storage Buffer:
  • 1X Sample Buffer (50 mM Tris-HCl, 2% SDS, 10% glycerol, 300 mM 2-mercaptoethanol, 0.01% Bromophenol blue)
  • Storage Instruction:
  • Store at -80°C. Aliquot to avoid repeated freezing and thawing.
  • Applications
  • Western Blot
  • Application Image
  • Western Blot
  • Gene Information
  • Entrez GeneID:
  • 3074
  • Protein Accession#:
  • -
  • Gene Name:
  • HEXB
  • Gene Alias:
  • ENC-1AS
  • Gene Description:
  • hexosaminidase B (beta polypeptide)
  • Gene Summary:
  • Hexosaminidase B is the beta subunit of the lysosomal enzyme beta-hexosaminidase that, together with the cofactor GM2 activator protein, catalyzes the degradation of the ganglioside GM2, and other molecules containing terminal N-acetyl hexosamines. Beta-hexosaminidase is composed of two subunits, alpha and beta, which are encoded by separate genes. Both beta-hexosaminidase alpha and beta subunits are members of family 20 of glycosyl hydrolases. Mutations in the alpha or beta subunit genes lead to an accumulation of GM2 ganglioside in neurons and neurodegenerative disorders termed the GM2 gangliosidoses. Beta subunit gene mutations lead to Sandhoff disease (GM2-gangliosidosis type II). [provided by RefSeq
  • Other Designations:
  • N-acetyl-beta-glucosaminidase,OTTHUMP00000128232,hexosaminidase B
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