ACP5 purified MaxPab mouse polyclonal antibody (B01P)
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More Files
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Specification
Product Description
Mouse polyclonal antibody raised against a full-length human ACP5 protein.
Immunogen
ACP5 (NP_001602.1, 1 a.a. ~ 325 a.a) full-length human protein.
Sequence
MDMWTALLILQALLLPSLADGATPALRFVAVGDWGGVPNAPFHTAREMANAKEIARTVQILGADFILSLGDNFYFTGVQDINDKRFQETFEDVFSDRSLRKVPWYVLAGNHDHLGNVSAQIAYSKISKRWNFPSPFYRLHFKIPQTNVSVAIFMLDTVTLCGNSDDFLSQQPERPRDVKLARTQLSWLKKQLAAAREDYVLVAGHYPVWSIAEHGPTHCLVKQLRPLLATYGVTAYLCGHDHNLQYLQDENGVGYVLSGAGNFMDPSKRHQRKVPNGYLRFHYGTEDSLGGFAYVEISSKEMTVTYIEASGKSLFKTRLPRRARP
Host
Mouse
Reactivity
Human
Quality Control Testing
Antibody reactive against mammalian transfected lysate.
Storage Buffer
In 1x PBS, pH 7.4
Storage Instruction
Store at -20°C or lower. Aliquot to avoid repeated freezing and thawing.
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Applications
Western Blot (Transfected lysate)
Western Blot analysis of ACP5 expression in transfected 293T cell line (H00000054-T02) by ACP5 MaxPab polyclonal antibody.
Lane 1: ACP5 transfected lysate(35.75 KDa).
Lane 2: Non-transfected lysate.
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Gene Info — ACP5
Entrez GeneID
54GeneBank Accession#
NM_001611.2Protein Accession#
NP_001602.1Gene Name
ACP5
Gene Alias
MGC117378, TRAP
Gene Description
acid phosphatase 5, tartrate resistant
Omim ID
171640Gene Ontology
HyperlinkGene Summary
This gene encodes an iron containing glycoprotein which catalyzes the conversion of orthophosphoric monoester to alcohol and orthophosphate. It is the most basic of the acid phosphatases and is the only form not inhibited by L(+)-tartrate. [provided by RefSeq
Other Designations
TrATPase|tartrate resistant acid phosphatase 5|tartrate-resistant acid ATPase
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Interactome
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Pathway
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Disease
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Publication Reference
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The effects of the cathepsin K inhibitor odanacatib on osteoclastic bone resorption and vesicular trafficking.
Leung P, Pickarski M, Zhuo Y, Masarachia PJ, Duong LT.
Bone 2011 Oct; 49(4):623.
Application:IF, Human, Osteoclasts.
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The effects of the cathepsin K inhibitor odanacatib on osteoclastic bone resorption and vesicular trafficking.
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