LGALS1 purified MaxPab rabbit polyclonal antibody (D01P)
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More Files
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Specification
Product Description
Rabbit polyclonal antibody raised against a full-length human LGALS1 protein.
Immunogen
LGALS1 (NP_002296.1, 1 a.a. ~ 135 a.a) full-length human protein.
Sequence
MACGLVASNLNLKPGECLRVRGEVAPDAKSFVLNLGKDSNNLCLHFNPRFNAHGDANTIVCNSKDGGAWGTEQREAVFPFQPGSVAEVCITFDQANLTVKLPDGYEFKFPNRLNLEAINYMAADGDFKIKCVAFD
Host
Rabbit
Reactivity
Human, Mouse
Quality Control Testing
Antibody reactive against mammalian transfected lysate.
Storage Buffer
In 1x PBS, pH 7.4
Storage Instruction
Store at -20°C or lower. Aliquot to avoid repeated freezing and thawing.
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Applications
Western Blot (Tissue lysate)
LGALS1 MaxPab rabbit polyclonal antibody. Western Blot analysis of LGALS1 expression in mouse spleen.Western Blot (Tissue lysate)
LGALS1 MaxPab rabbit polyclonal antibody. Western Blot analysis of LGALS1 expression in human kidney.Western Blot (Cell lysate)
LGALS1 MaxPab rabbit polyclonal antibody. Western Blot analysis of LGALS1 expression in HeLa.Western Blot (Transfected lysate)
Western Blot analysis of LGALS1 expression in transfected 293T cell line (H00003956-T01) by LGALS1 MaxPab polyclonal antibody.
Lane 1: LGALS1 transfected lysate(14.70 KDa).
Lane 2: Non-transfected lysate.
Immunofluorescence
Immunofluorescence of purified MaxPab antibody to LGALS1 on HeLa cell. [antibody concentration 10 ug/ml] -
Gene Info — LGALS1
Entrez GeneID
3956GeneBank Accession#
NM_002305Protein Accession#
NP_002296.1Gene Name
LGALS1
Gene Alias
DKFZp686E23103, GAL1, GBP
Gene Description
lectin, galactoside-binding, soluble, 1
Omim ID
150570Gene Ontology
HyperlinkGene Summary
The galectins are a family of beta-galactoside-binding proteins implicated in modulating cell-cell and cell-matrix interactions. This gene product may act as an autocrine negative growth factor that regulates cell proliferation. [provided by RefSeq
Other Designations
OTTHUMP00000028982|S-Lac lectin 1|beta-galactoside-binding lectin L-14-I|galaptin|galectin 1|galectin-1|lactose-binding lectin 1
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Interactome
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Disease
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Publication Reference
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Transgenic overexpression of the α7 integrin reduces muscle pathology and improves viability in the dy(W) mouse model of merosin-deficient congenital muscular dystrophy type 1A.
Doe JA, Wuebbles RD, Allred ET, Rooney JE, Elorza M, Burkin DJ.
J Cell Sci 2011 Jun; 124:2287.
Application:IF, IHC-Fr, WB-Ti, Mouse, Gastrocnemius muscles, Tibialis anterior muscles.
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Transgenic overexpression of the α7 integrin reduces muscle pathology and improves viability in the dy(W) mouse model of merosin-deficient congenital muscular dystrophy type 1A.
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